Interventions (JMJD3)

London Futurists - Interventions to Trigger “Younging” Mechanisms: www.youtube.com/watch?v=mghDE-KWdeA

NameDescription
M1 phenotypeInflammatory macrophage
M2 phenotypeAnti-inflammatory macrophage
MicrogliaBrain macrophages
CAPCholinergic anti-inflammatory pathway in the brain and spleen (acetylcholine) developed 150 million years ago to reduce circulating inflammation.

FactorFunction
NAD and sirtuinsHistone acetylation and methylation play important roles in longevity-related metabolic pathways
KlothoPromoter region demethylation necessary for longevity benefits
Hypoxia / oxygen therapyVarioxia leads to JMJD3 expression
Rapamycin / mTORmTOR pathway histone methylation patterns implicated in gliablastoma tumor cell proliferation
Integrated stress response (hypoxia, low amino acids)ATF4 triggers JMJD3 expression
Vitamin D3 pathwayActivates JMJD3
Blood borne longevity factorsVarioxia leads to JMJD3 expression
Blood factor dilution or addition rejuvenationImplicates H3K27me3 reduction and CAP
Yamanaka factors (OSKM) epigenome reprogrammingAll OSKM factors implicate JMJD3 / H3K27
Alpha-ketoglutarate (AKG) supplementationIncreases JMJD3
NRF2Copromoted along with JMJD3
AutophagyJMJD3 regulates expression of autophagy genes
FastingInduces JMJD3
Heterochronic parabiosisInduces JMJD3
REM sleepepigenetic health

ProteinDescription
JMJD3 (Jumonji domain-containing protein-3)Epigenetic regulating: histone demethylase regulating H3K27me3
H3K27me3Trimethylation of histone H3 on lysine 27